<bold>

Bold

Used to mark text that should appear in bold face.

Remarks

Emphasis as a Toggle Switch: The @toggle attribute controls the behavior of this element. When the value of @toggle is set to “no”, the emphasized text remains in the requested style, no matter what the surrounding text does. When the value of @toggle is “yes”, if the surrounding text is set to the same emphasis style, the text within this element will change to another emphasis style, so that the text will always be typographically distinct from its surroundings.
Using the element <italic> as an example, setting the @toggle attribute to “no” would mean that a material marked as italics will always be italics, even in an italic context. In contrast, if the @toggle attribute was set to “yes” on the <italic> element, if the formatting context imposes italics (whether due to another <italic> element, a stylesheet, some CSS, or other means), then the italics would be turned off within that context, making the emphasized text emphasized by contrast, but not italic. The <italic> element would still produce italics everywhere else.

Attributes

id Document Internal Identifier
specific-use Specific Use
toggle Toggle Switch
xml:base Base

Content Model

<!ELEMENT  bold         (#PCDATA %emphasized-text;)*                 >

Expanded Content Model

(#PCDATA | email | ext-link | uri | inline-supplementary-material | related-article | related-object | bold | fixed-case | italic | monospace | overline | roman | sans-serif | sc | strike | underline | ruby | alternatives | inline-graphic | private-char | chem-struct | inline-formula | tex-math | mml:math | abbrev | milestone-end | milestone-start | named-content | styled-content | fn | target | xref | sub | sup)*

Description

Any combination of:

This element may be contained in:

<addr-line>, <aff>, <alt-title>, <article-title>, <attrib>, <award-id>, <bold>, <chapter-title>, <chem-struct>, <code>, <collab>, <comment>, <compound-kwd-part>, <compound-subject-part>, <conf-theme>, <copyright-statement>, <corresp>, <data-title>, <def-head>, <disp-formula>, <element-citation>, <ext-link>, <fixed-case>, <funding-source>, <funding-statement>, <gov>, <inline-formula>, <inline-supplementary-material>, <italic>, <kwd>, <label>, <license-p>, <meta-value>, <mixed-citation>, <monospace>, <named-content>, <on-behalf-of>, <overline>, <p>, <part-title>, <preformat>, <price>, <product>, <rb>, <related-article>, <related-object>, <role>, <roman>, <sans-serif>, <sc>, <series>, <series-text>, <series-title>, <sig>, <sig-block>, <source>, <std>, <strike>, <styled-content>, <sub>, <subject>, <subtitle>, <sup>, <supplement>, <target>, <td>, <term>, <term-head>, <textual-form>, <th>, <title>, <trans-source>, <trans-subtitle>, <trans-title>, <underline>, <verse-line>, <xref>

Example 1

...
<p>... The third nucleotide (CG<bold>A</bold>) at codon 529, which
is a specific nucleotide of <italic>M. tuberculosis</italic>
(<xref ref-type="bibr" rid="B8">8</xref>, <xref ref-type="bibr"
rid="B11">11</xref>), was not clearly determined by conventional PCR direct
sequencing. Furthermore, PCR sequencing misidentified codon 531 as
T<bold>C</bold>G, which reflects the rifampin-susceptible strain. However,
the second nucleotide at codon 531, which is the most frequent site of
mutation related to rifampin resistance, was determined as
T<bold>T</bold>G by nested PCR sequencing (Fig. <xref ref-type="fig"
rid="F5">5</xref>, lower panel). ...</p>
...

Example 2

...
<table frame="box" rules="all" cellpadding="5">
<thead>
<tr>
<td>Functional Category</td>
<td>Gene or Operon</td>
<td>&sigma; Factor<sup>a</sup></td>
<td>Activated by<sup>b</sup></td>
<td>Repressed by</td>
</tr>
</thead>
<tbody>
<tr>
<td>Engulfment</td>
<td><italic>spoIID</italic></td>
<td><bold>&sigma;<sup>E</sup></bold></td>
<td></td>
<td><bold>SpoIIID</bold></td>
</tr>
<tr>
<td></td>
<td><italic>spoIIM</italic></td>
<td><bold>&sigma;<sup>E</sup></bold></td>
<td></td>
<td>GerR</td>
</tr>
...</tbody>
</table>
...

Journal Publishing Tag Library NISO JATS Draft Version 1.1d3

Version of April 2015

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