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<bold> Bold

Used to mark text that should appear in bold face.

Usage/Remarks

Ensuring that a text component remains bold is not as common in journal publishing as (for example) italics, since bold is typically reserved for headings. But any text may be marked as bold and kept as bold or toggled, using the @toggle attribute.
Using the Toggle Switch with Bold
The @toggle attribute can control the behavior of the <bold> element. For example, setting the @toggle attribute to “no” would mean that material marked as bold will always be bold, even in a context such as a bold heading. In contrast, if the @toggle attribute is set to “yes” on a <bold> element, and if the formatting context then imposes bold (whether due to another <bold> element, a stylesheet, some CSS, or other means), then the bolding would be turned off within that context, making the emphasized text emphasized by contrast (typographically distinct from its surroundings) but no longer bold.
Attributes
specific-use
toggle

Multi-lang Attributes

lang-focus
lang-focus-custom
lang-group
lang-source
lang-source-custom
lang-translate
lang-variant
lang-variant-custom

Base Attributes

id
xml:base
xml:lang
Models and Context
May be contained in
<abbrev>, <abbrev-journal-title>, <addr-line>, <aff>, <alt-title>, <anonymous>, <article-title>, <attrib>, <award-id>, <bold>, <chapter-title>, <chem-struct>, <code>, <collab>, <collab-name>, <comment>, <compound-kwd-part>, <compound-subject-part>, <conf-acronym>, <conf-date>, <conf-loc>, <conf-name>, <conf-num>, <conf-sponsor>, <conf-theme>, <copyright-statement>, <corresp>, <data-title>, <date-in-citation>, <def-head>, <degrees>, <disp-formula>, <edition>, <element-citation>, <email>, <etal>, <ext-link>, <fax>, <fixed-case>, <funding-source>, <funding-statement>, <given-names>, <gov>, <history>, <inline-formula>, <inline-media>, <inline-supplementary-material>, <institution>, <issue>, <issue-part>, <issue-sponsor>, <issue-subtitle>, <issue-title>, <italic>, <journal-subtitle>, <journal-title>, <kwd>, <label>, <license-p>, <meta-name>, <meta-value>, <mixed-citation>, <monospace>, <named-content>, <on-behalf-of>, <overline>, <p>, <part-title>, <patent>, <phone>, <prefix>, <preformat>, <price>, <product>, <publisher-loc>, <publisher-name>, <rb>, <related-article>, <related-object>, <resource-name>, <role>, <roman>, <sans-serif>, <sc>, <see>, <see-also>, <self-uri>, <series>, <series-text>, <series-title>, <sig>, <sig-block>, <size>, <source>, <speaker>, <std>, <std-organization>, <strike>, <string-conf>, <string-date>, <string-name>, <styled-content>, <sub>, <subject>, <subtitle>, <suffix>, <sup>, <supplement>, <support-source>, <surname>, <target>, <td>, <term>, <term-head>, <textual-form>, <th>, <title>, <trans-source>, <trans-subtitle>, <trans-title>, <underline>, <unstructured-kwd-group>, <uri>, <verse-line>, <version>, <volume>, <volume-id>, <volume-series>, <x>, <xref>
Description
Any combination of:
Content Model
<!ELEMENT  bold         (#PCDATA %emphasized-text;)*                 >
Expanded Content Model

(#PCDATA | email | ext-link | uri | inline-supplementary-material | related-article | related-object | hr | bold | fixed-case | italic | monospace | overline | overline-start | overline-end | roman | sans-serif | sc | strike | underline | underline-start | underline-end | ruby | alternatives | inline-graphic | inline-media | private-char | chem-struct | inline-formula | tex-math | mml:math | abbrev | index-term | index-term-range-end | milestone-end | milestone-start | named-content | styled-content | fn | target | xref | sub | sup | x | break)*

Tagged Samples
Highlighting in text 
...
<p>... The third nucleotide (CG<bold>A</bold>) at codon 529, which 
 is a specific nucleotide of <italic>M. tuberculosis</italic>
 (<xref ref-type="bibr" rid="B8">8</xref>, <xref ref-type="bibr"
 rid="B11">11</xref>), was not clearly determined by conventional PCR direct
 sequencing. Furthermore, PCR sequencing misidentified codon 531 as
 T<bold>C</bold>G, which reflects the rifampin-susceptible strain. However,
 the second nucleotide at codon 531, which is the most frequent site of
 mutation related to rifampin resistance, was determined as
 T<bold>T</bold>G by nested PCR sequencing (Fig. <xref ref-type="fig"
 rid="F5">5</xref>, lower panel). ...</p>
...
Highlighting in table cell 
...
<table frame="box" rules="all" cellpadding="5">
 <thead>
  <tr>
   <td>Functional Category</td>
   <td>Gene or Operon</td>
   <td>&sigma; Factor<sup>a</sup></td>
   <td>Activated by<sup>b</sup></td>
   <td>Repressed by</td>
  </tr>
 </thead>
 <tbody>
  <tr>
   <td>Engulfment</td>
   <td><italic>spoIID</italic></td>
   <td><bold>&sigma;<sup>E</sup></bold></td>
   <td></td>
   <td><bold>SpoIIID</bold></td>
  </tr>
  <tr>
   <td></td>
   <td><italic>spoIIM</italic></td>
   <td><bold>&sigma;<sup>E</sup></bold></td>
   <td></td>
   <td>GerR</td>
  </tr>
  ...
 </tbody>
</table>
...